Mental Health Phenotypes of well-controlled HIV in Uganda

January 27, 2025 by
Mental Health Phenotypes of well-controlled HIV in Uganda
Aber Maurine
Leah H. Rubin1,2,3,4*, Kyu Cho5, Jacob Bolzenius5, Julie Mannarino5, Rebecca E. Easter1, Raha M. Dastgheyb1, Aggrey Anok6, Stephen Tomusange6, Deanna Saylor1, Maria J. Wawer4, Noeline Nakasujja7, Gertrude Nakigozi6, Robert Paul5*

Affiliations:

1Department of Neurology, Johns Hopkins University School of Medicine, Baltimore, MD, United States
2Department of Psychiatry and Behavioral Sciences, Johns Hopkins University School of Medicine, Baltimore, MD, United States
3Department of Molecular and Comparative Pathobiology, Johns Hopkins University School of Medicine, Baltimore, MD, United States
4Department of Epidemiology, Johns Hopkins Bloomberg School of Public Health, Baltimore, MD, United States
5Missouri Institute of Mental Health, University of Missouri - St. Louis, St. Louis, MO, United States
6Rakai Health Sciences Program, Kalisizo, Uganda
7Department of Psychiatry, Makerere University, Kampala, Uganda

*Corresponding authors: lrubin@jhmi.edu; robert.paul@umsl.edu


ABSTRACT

Introduction

The phenotypic expression of mental health (MH) conditions among people with HIV (PWH) in Uganda and worldwide are heterogeneous.Accordingly, there has been a shift toward identifying MH phenotypes usingdata-driven methods capable of identifying novel insights into mechanisms of divergent MH phenotypes among PWH. We leverage the analytic strengths of machine learning combined with inferential methods to identify novel MH phenotypes among PWH and the underlying explanatory features.


Methods

A total of 277 PWH (46% female, median age = 44; 93% virally suppressed [<50copies/mL]) were included in the analyses. Participants completed the Patient Health Questionnaire (PHQ-9), Beck Anxiety Inventory (BAI), and the PTSD Checklist-Civilian (PCL-C). A clustering pipeline consisting of dimension reduction with UMAP followed by HBDScan was used to identify MH subtypes using total symptom scores. Inferential statistics compared select demographic (age, sex, education), viral load, and early life adversity between clusters.


Results

We identified four MH phenotypes. Cluster 1 (n = 76; PTSD phenotype) endorsed clinically significant PTSD symptoms (average PCL-C total score > 33). Clusters 2 (n = 32; anxiety phenotype) and 3 (n = 130; mixed anxiety/depression phenotype) reported minimal PTSD symptoms, with modest BAI (Cluster 2) and PHQ-9 (Cluster 3) elevations. Cluster 4 (n = 39; minimal symptom phenotype) reported no clinical MH symptom elevations. Comparisons revealed higher rates of sexual abuse during childhood among the PTSD phenotype vs. the minimal symptom phenotype (p = 0.03).


Discussion 

We identified unique MH phenotypes among PWH and confirmed the importance of early life adversity as an early risk determinant for unfavorable MH among PWH in adulthood.


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